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Enter the breakable filament model (see for instance [Knowles 2009, Aguzzi & Falsig 2012]): We know that this is how it works for the [PSI + ] prion in yeast, with the chaperone Hsp104 responsible for doing the fragmenting [Chernoff 1995] — see this backgrounder

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Why researchers pair fat-metabolism peptides with growth-hormone-secretagogue peptides Growth-hormone secretagogues are studied for their role in stimulating endogenous growth hormone release, which is linked in the literature to both lipolytic activity and protein synthesis pathways making them a natural pairing candidate for researchers examining fat metabolism and lean tissue outcomes within the same study design

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